Close
  Indian J Med Microbiol
 

Figure 2: (a) Transmission electron microscopic examination confirms presence of abundant crystalline structures in PTE (transmission electron microscopy × 1050); (b) Crystals spilling over into the lumen of proximal convoluted tubules with destruction of the lining epithelial cells (transmission electron microscopy × 1250); (c) Interstitial histiocytes brimming with crystals (transmission electron microscopy × 1250); (d) Localisation of crystals in cytoplasm of interstitial histiocytes (transmission electron microscopy × 4200); (e) Damaged podocyte bodies containing crystalline inclusions (transmission electron microscopy × 2550); (f) Crystals in cytoplasm of an injured podocyte with degenerative changes (transmission electron microscopy × 8200); (g) High magnification displays fine crystal lattice substructure (transmission electron microscopy × 20,500)

Figure 2: (a) Transmission electron microscopic examination confirms presence of abundant crystalline structures in PTE (transmission electron microscopy × 1050); (b) Crystals spilling over into the lumen of proximal convoluted tubules with destruction of the lining epithelial cells (transmission electron microscopy × 1250); (c) Interstitial histiocytes brimming with crystals (transmission electron microscopy × 1250); (d) Localisation of crystals in cytoplasm of interstitial histiocytes (transmission electron microscopy × 4200); (e) Damaged podocyte bodies containing crystalline inclusions (transmission electron microscopy × 2550); (f) Crystals in cytoplasm of an injured podocyte with degenerative changes (transmission electron microscopy × 8200); (g) High magnification displays fine crystal lattice substructure (transmission electron microscopy × 20,500)